Dr. James Schaller, MD
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Bartonella Henselea a Cause of Major Disease and Illness

THREE YEARS OF TREATMENT FAILS?
Bartonella Henselae as Cause of Serious Fatigue, Depression, Agitation, Memory Loss, Joint Pain, Headaches, Major Neurology Disease, Severe Destructive Inflammation and Guillain-Barre

Three family members were bitten by an insect and developed mild, moderate and very severe neurology and psychiatric disease. Over the ensuing months, the youngest son was diagnosed with Guillain-Barre syndrome followed by Chronic Inflammatory Demyelinating Polyradiculoneuropathy (CIDP). The older son developed intermittent disorientation and irritability, and the mother experienced fatigue, headaches, joint pain and memory loss.

When tested approximately three years after the woodlouse hunter spider infestation, all three family members were Bartonella henselae seroreactive and B. henselae DNA positive by amplified and sequenced sources from blood, serum or Bartonella enrichment blood cultures uniquely developed by the authors, Also, B. henselae DNA was found in two insects near the home. Specifically, Bartonella was found in a woodlouse and woodlouse hunter spiders collected adjacent to the family's home. The spider was the suspected case of transmission.

It should be noted that in contrast to the belief Bartonella is easy to remove, which I opposed over six years ago, some still cling to the idea it is short-lived and easy to kill, but all failed to clear the infection. Of course, a desperate position would be to say they were re-infected in the weeks prior to the follow up testing. Since infection societies are so backward and dated to not even offer guidelines that mention Bartonella, and prove by many advanced direct and indirect means cure of Bartonella, we will still see this infection treated with tired, dated and failing treatments in traditional and alternative medicine approaches.

Further, the symptoms of these patients are symptoms usually attributed to many psychiatric disorders, or Lyme and Babesia infections. I doubt 1/100 using common diagnostic techniques would have believed these symptoms were due to Bartonella, since that merely causes “cat scratch fever and is easily killed, supposedly, by a series of unique herbs, tinctures, quinolones like Cipro or Levaquin, azithromycin, mycobutin, rifampin, minocycline, clarithromycin and a series of seasonal new proposals. None seem to be based on direct and indirect testing. First, because they do not have a license to test with such testing because they are not physicians, because they read so little or trust educational Popes who do not modify their position in emerging infections in any real and meaningful manner over years or even a decade.

It is possible despite the bite marks, that we do not know the source of these infections. However, how could any physician ignore or minimize an infection that could cause or contribute to Guillain-Barre syndrome, Chronic Inflammatory Demyelinating Polyradiculoneuropathy (CIDP), intermittent disorientation, irritability, fatigue, headaches, joint pain and memory loss found in any patient they valued? It is hardly new, if one reads the world medical literature fully, that this brilliant team of researchers is proposing findings outside of Bartonella’s range of human body damage. The proposed findings are well within the proposals of the extensive literature with their helpful additions.

In conclusion, it should be noted that insect infections are proposed as a possible source for serious and moderate disease. This is not generally accepted because few have taken the time to self-educate as is part of all medical school and residency training, and read a mere summary and feel certain about these vector infections. It takes years to read thousands of articles around these emerging infections to begin to have any mastery, and lazy appeals to the top ten studies, is simply using statistics to save the intellectual sweat.

One family member has CIDP which is a serious nerve disease. A simple definition might include this information below:

The key to this disease is loss of the highly critical fatty myelin sheath (the fatty covering that insulates, protects and is critical to nerve function) of the nerves outside the brain and spine.

Chronic inflammatory demyelinating polyneuropathy is believed to be due to immune cells, cells which normally protect the body from foreign infection, but which turn on the human patient, and incorrectly attack their body nerves. The impact of this self-attack is some nerves do not work at all or only slightly. Others experience numbing, tingling, pain, muscle weakness, loss of reflexes, along with abnormal sensations and fatigue. It often gets worse over time.

CIDP diagnosis is very hard, and it is impressive this lab was able to determine this finding. Some suggest it is routinely missed and under-treated in part because it has many different types or different symptoms clusters. Or, as in so many other medical areas, the lab testing, history, physical exam findings and electrical test criteria are variable and/or insensitive according to a modified entry in Wikipedia.

Modified source: www.parasitesandvectors.com

Bartonella henselae infection in a family experiencing neurological and neurocognitive abnormalities after woodlouse hunter spider bites
PE Mascarelli, RG Maggi, S Hopkins, B R Mozayeni, CLTrull, JM Bradley, BC Hegarty and EB Breitschwerdt.




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