Dr James Schaller
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Lyme affects the brain in 10 days and creates a rigid, unmotivated float type personality. Smart people put the car, the kids, or painting a room before getting Lyme IgeneX tests. Individuals who are usually insightful just stop thinking about the fact that a Lyme spirochete moving unchecked in their body is not something to put number 10 on the "to do" list, or next month in the daily planner!

Lyme inflammation chemicals quickly affect the frontal brain lobes, which are used in advanced and subtle self-reflection. In the last 5 years, I know of many who have ignored me and are now buried or have buried loved ones.

Some folks would rather be right than save their health and the health of their loved ones. They live in the 1950s, where Dr. Marcus Welby always knew everything in his field, and since he was nice, he was never wrong. Now we live in a world where good ole Marcus is unable to read 5 journals a month in his limited specialty, and medical references in one database exceed 11 million. And I think much of the best cutting-edge, non-surgical medicine is not even published yet.

Lyme disease can mimic many other real illnesses, such as arthritis, Alzheimer's, fibromyalgia, lupus, chronic fatigue, mononucleosis, multiple sclerosis, etc. Unless a physician has accurate, up-to-date information, it is very likely they will miss Lyme. If they have not used IgeneX laboratory to do the testing, which is never at a routine local lab, any Lyme testing done was junk.

Much to my surprise, lab results and clinical symptoms which I was taught to believe showed arthritis, Alzheimer's, fibromyalgia, lupus, chronic fatigue, mononucleosis and multiple sclerosis, I am finding in very clearly diagnosed Lyme patients. Patients then improve with aggressive antibiotic treatment. Do you understand? These illnesses are not supposed to improve with Lyme killing antibiotics! It has stunned me to see this at times. This misdiagnosis problem is routinely and relentlessly committed by physicians who are much smarter than me and work at big name hospitals all over the US.

If I had a dollar for every person who has said to me, "James, I know you mean well, but you just need to understand. I was treated at the prestigious Super Big Name Hospital by the head physician whose name is Dr. Superman, who is regarded as the great deity of my illness all over the galaxy. Of course if I had Lyme this great man of medicine could never have missed it."

Guess what, I know people that have buried loved ones and become profoundly ill who have said this to me. Even great doctors can have blind spots big enough to drive a hummer through. And I have found illnesses and created new treatments that they have missed. Sincerity does not equal thoroughness.

Bluntly, since Lyme is the number one vector illness in the US, all of these disorders should probably only be considered after a clear negative result from IgeneX Laboratories in which the Jones Western Blot criteria are used (See: Approach to Reading Western Blots on this site.)


Title: The Pathologist's View of Lyme disease

Authors: Hejka, England, Schmitz, Schell--Madison
Source: Wisconsin Medical Journal 1989;88(11):17-20

"Lyme arthritis is FREQUENTLY mistaken clinically for rheumatoid arthritis, systemic lupus erythematous, or Reiter's syndrome. Synovial biopsies are probably the most frequent tissue examined microscopically from patients with Lyme disease. The histopathological spectrum ranges from chronic hyperplastic synovitis with effusions to erosive ostearthritis. (20)... "The disease captures the imagination of physicians, especially in endemic areas such as Wisconsin. Lyme disease is a multisystem disorder with protean manifestations ..."

Definite Lyme disease instead of lupus:

Nancy Brown, lbrown@snowcrest.net, Weaverville, Northern CA

"Following a long-standing, untreated chronic borreliosis, I, too, eventually developed anti DS-DNA antibodies and a positive ANA, along with other markers for Lupus. However, the late Paul Lavoie had numerous patients with similar laboratory results, all of which returned to normal or significantly improved with long-term antibiotic therapy. He firmly believed that in some people, the Lupus or Lupus-type syndrome was a reaction to the underlying Lyme disease infection. By treating the Lyme disease infection, the auto-immune attack often backs off. In fact, he had numerous patients in which that exact thing happened as a result of long-term antibiotic therapy.

I am one of those patients. While being treated with anti-inflammatory medications, etc. for a condition "highly suggestive of systemic lupus erythematosis" by a rheumatologist, my auto-immune laboratory tests continued to go higher and higher. After seeing Dr. Lavoie and undergoing long-term antibiotic therapy (initially with no other change in therapy) all the Lupus tests gradually improved, directly paralleling the administration of antibiotics.

It was touch and go for awhile handling the side effects of the antibiotic therapy; trying to determine if the flare in symptoms was a "Lupus flare" resulting from the antibiotics (many Lupus patients attest to this occurence) or a herxheimer-like response from dealing with the Lyme disease infection.

The bottom line is that because of his compassionate therapy, individualized to my needs, I improved tremendously, all the Lupus laboratory tests returned to 100% NORMAL, and I have returned to a reasonably productive life (not cured; still experiencing symptoms on a much milder level) without any medications other than an occasional ibuprofen for almost 3 years now."

See complete posting at: sci.med.diseases.lyme: Re: Lyme & Lupus groups.google.com


by Andrea Stanton
Published in the Plain Dealer, Gloucester County, NJ edition of August 7, 1997

Lois Ravotti is a Williamstown resident and has had Lyme disease for over 20 years. In the late 1970's, Ravotti enjoyed camping and attending events such as the Philadelphia Folk Festival. She traces her infection to a camping trip to Bass River State Forest, which has turned out to be a hot spot for disease - carrying ticks, according to Ravotti. Over the years, she has been misdiagnosed many times. Lyme disease mimics other illnesses. One doctor told her that she had lupus. Since Lyme disease was not as prevalent years ago and many doctors were unfamiliar with the illness, many sufferers did not get medical treatment in the crucial early stages of the sickness. Ravotti was one of them. In January of 1996, she finally was diagnosed with the illness. Like Marge, Ravotti has always tested negative on her Lyme test.


By John D. Bleiweiss (deceased), MD Trenton, NJ. 4/94

"Prior to proper diagnosis, patients habitually report that they were assigned the following diagnoses most often: Chronic Fatigue Syndrome, Multiple Sclerosis, Fibromyalgia, Lupus, Candidiasis, Chronic Mononucleosis, Hypoglycemia and Stress-related illness. If these appear in a differential diagnosis, then Lyme Disease (LD) should also be considered."


"[For Lyme disease] Very often, the pinna and ear lobes are varying shades of red. Less commonly, a similar erythema can be observed on the hands or malar (upper cheeks) areas. A malar rash is not pathognomonic of Lupus, if in fact SLE is distinct from LD (Abstract 55A, V LD Symposium)."


"Dr. Lavoie [also has died]has published the coincident findings of LD with RA, and SLE (lupus) with LD. The SLE was associated with positive DS-DNA (double stranded DNA) which is considered diagnositic for lupus. This marker improved with antibiotic treatment for the LD. The author felt that the LD might be causing/aggravating the SLE."

See complete article at: www.lymealliance.org/html/med5.html

Media articles about Lyme disease and lupus

Jodi Norton: Adversity Doesn't Keep Diver from Soaring.

Columbia University Record, October 10, 1997

"Lupus is an incurable, non-contagious disease that strikes the auto-immune system and can be deadly. In addition to enduring the chronic pain caused by Lupus, Norton's diving season and school work were interrupted many times by trips to the emergency room with meningitis, pericarditis (inflammation of the sac around the heart) and swelling of tissue around the brain. She also has been battling Lyme's [sic] Disease."



"I am a 31 year old mom of a 13 month old little boy. I was recently diagnosed with Lyme. Only after 10 years of a mysterious "illness". ... I was misdiagnosed with mitral valve prolapse, irritable bowel syndrome, interstitial cystitis, and for the last 5 years I have supposedly had Lupus."

See complete posting at: sci.med.diseases.lyme: congenital lyme concerns


Martha Behrle mrsmeister@aol.com northern NJ

"I know of several people with Lyme who were intitally diagnosed with Lupus due to their elevated ANA count... my ANA was elevated at one point during my treatment and I also early on had the Lupus "butterfly" rash across my face ..."

See complete posting at: sci.med.diseases.lyme: Re: lyme disease causing false postitive ana for lupus?


Clinical pathologic correlations of Lyme disease by stage.

AUTHORS: Duray PH; Steere AC
AUTHOR AFFILIATION: Department of Pathology, Fox Chase Cancer Center, Philadelphia, Pennsylvania 19111.
SOURCE: Ann N Y Acad Sci 1988;539:65-79
CITATION IDS: PMID: 2847622 UI: 89048814

Lyme disease is capable of producing a wide variety of clinical pathologic conditions and lesions having in common histologic features of collagen-vascular disease. The plasma cell is an omnipotent inflammatory responder in most tissues involved by Lyme disease, ranging from relatively acute to lesions that have gone on for years. Vascular thickening also seems to be prominent, and in the dermis is accompanied by scleroderma-like collagen expansion.

The disease in some ways resembles the responses seen in lupus erythematosus such as mild cerebritis with lymphocytes and plasma cells in the leptomeninges. Lymphoplasmacytic panniculitis of Lyme disease resembles lupus profundus, both in the infiltrate and the plasma cell-blood vessel relationship. The onion skin thickened vessels of the synovia resemble the vessels of lupus spleens, while the scleradermoid thickening of the dermis and various skin lesions of stage III Lyme disease suggest a collagen-vascular disorder. Finally, the perivascular lymphoid infiltrate in clinical myositis does not differ from that seen in polymyositis or dermatomyositis. All of these histologic derangements suggest immunologic damage in response to persistence of the spirochete, however few in number.



FOR MORE COMPLETE FURTHER INFORMATION GO TO: https://www.geocities.com/lymeart3/lupus.txt

To Your Health,

Dr. J

Bank Towers, Tamiami Trail, Naples, FL
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