Lyme disease (LD) can affect the skin, joints, heart, and nervous system as a multisystemic condition. The cause of the illness is the spirochete of the genus Borrelia. These pathogens can affect the skin, joints, heart, and nervous system.
Lyme neuroborreliosis (LNB) is the term for the disease that occurs when the nervous system is involved. [The CDC feels Lyme is in the brain in 12 days. The point being Lyme loves fat, and neurons are fatty, and the brain gets about 20% of your blood.]
The most significant change… is inflammation of the central nervous system (CNS, or brain) and the peripheral nervous system (PNS). Furthermore, clinical features of neurological manifestations, such as meningitis, encephalitis, radiculopathies, and cranial neuritis. [I have found over 25 cognitive neurology signs by interview and special brain mentation scales]
However, dementia-like syndrome is an infrequent manifestation of Lyme disease. Maybe. [A MacDonald found it in the brains of seven or 12 Harvard brain bank Altzeimer’s patients, and Embers found it in the nerves of an overtreated patient who should have been cured].
Cases of secondary dementia syndrome due to nervous system involvement, as well as post-treatment, have been described. A thorough examination, medical history, laboratory and imaging studies, cerebrospinal fluid (CSF) examination, MRI…
and fludeoxyglucose-18-positron emission tomography (FGD-PET) are required to differentiate between these syndromes.
I cannot recall if more than 10% of neurologists got a nuclear PET scan that shows the metabolism of every cm of the brain. I ordered the last one for a patient with severe cognitive loss. The neurologist was very busy, and I am not sure he noticed cognitive changes.