SIMPLE BASICS TO HELP YOUR IMMUNE SYSTEM
KILL LYME DISEASE, BARTONELLA AND BABESIA
SIMPLE BASICS TO HELP YOUR IMMUNE SYSTEM
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Babesia
The Signs and Symptoms of Babesia
# Sign or Symptom Full Reference 1 Fever Vannier E, Krause PJ. Human babesiosis. N Engl JMed. 2012;366(25):2397-2407. doi:10.1056/NEJMra1202018 2 Chills Vannier E, Krause PJ. Human babesiosis. N Engl JMed. 2012;366(25):2397-2407. doi:10.1056/NEJMra1202018 3 Sweats Vannier E, Krause PJ. Human babesiosis. N Engl JMed. 2012;366(25):2397-2407. doi:10.1056/NEJMra1202018 4 Fatigue Vannier E, Krause PJ. Human babesiosis. N Engl JMed. 2012;366(25):2397-2407. doi:10.1056/NEJMra1202018 5 Malaise
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Bartonella
The Main Bartonella Signs and Symptoms
The frequency of some of these is flawed. Lymph node symptoms are only about 1-3%, and it can go away despite ongoing infection. The reporting that mental symptoms are rare is wrong. I published some in 2007, and in recent years, Breitschwerdt, Maggi, and their team and associates have published extensive psychiatric, neuropsychology, and neurology suffering. # Symptom / Sign
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Lyme Disease
Lyme Disease’s 110 Signs and Symptoms
110 signs/symptoms table # Sign or Symptom Typical Frequency Reference 1 Erythema migrans rash Common Steere AC, et al. N Engl J Med. 2001;345(2):115-125. 2 Expanding rash Common Wormser GP, et al. Clin Infect Dis. 2006;43(9):1089-1134. 3 Central clearing rash Common Hu L. Ann Intern Med. 2016;164(9):ITC65-ITC80. 4 Warmth at rash site Common CDC. Signs and Symptoms of Untreated Lyme
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Lyme Disease
Top 80 Documented Signs and Symptoms of Lyme Disease
1. Erythema Migrans Rash Reference: Steere AC, et al. The clinical evolution of Lyme arthritis. Ann Intern Med. 1983;99(1):76-82. Reference: Wormser GP, et al. The clinical assessment, treatment, and prevention of Lyme disease. Clin Infect Dis. 2006;43(9):1089-1134. 2. Expanding Annular Rash Reference: Steere AC, et al. Lyme disease. N Engl J Med. 2001;345(2):115-125. 3. Central Clearing in Rash Reference: Hu
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Uncategorized
50 Borrelia Debris / Inflammatory-Particle Master Table
The strongest direct persistence evidence is for peptidoglycan in human Lyme arthritis, cartilage-associated antigens in treated mice, and DNA/mRNA persistence after lethal ceftriaxone exposure in vitro. Evidence Key H = human evidence; M = mammal/animal; V = in vitro. Persistence: ★★★ direct post-treatment/post-killing evidence; ★★ suggestive/indirect; ★ component established but persistence itself unproven. # Particle / Borrelia Material Persistence /
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Uncategorized
Morgellons, Fibers, Specks, Lesions and Psychiatry
In 100% of my decades of Morgellons patients I have found multiple infections. Never one type. These all have impact on the skin and head tissues. And they are in thousands of miles of brain vessels. Sadly, most doctors have a full waiting room and a rapid mill. The problem this creates is Morgellons patients are distressed, agitated, overwhelmed and
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Babesia, Bartonella, Lyme Disease
Top Expert Bartonella Doctor Reports It Took 81 Days to Get a Positive Antibody Test
I have repeatedly tried to warn Lyme-literate healers that antibody tests are often negative without treatment. The treatment kills Bartonella, Babesia, and Lyme so you are not undertreated years, and get ill. Since almost all our patients have been previously treated with Lyme-literate doctors it is sad to see so many patients wasting time, money, and function due to ignorance
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Lyme Disease
New Study Shows Standard Lyme Tests Miss Most Early Infections
A newly published study in the Journal of Clinical Microbiology puts concrete data behind a long recognized problem in Lyme disease care. The research, supported by the Bay Area Lyme Foundation and based on data from the Lyme Disease Biobank, found that FDA-cleared testing methods miss between 64 and 78 percent of early Lyme cases. This includes patients who present
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Lyme Disease
50 Things That Persist After Lyme Germs Are Destroyed by Herbs or Volatile Oils
Below is a 50-item Borrelia debris / inflammatory-particle master list. I am separating two different scientific questions: A. Has the material actually been shown to persist after Borrelia loses viability or after antibiotic treatment? B. Is the molecule a Borrelia component capable of provoking inflammation if released as debris? Only a subset has strong direct evidence for post-killing persistence, particularly
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