BABESIA, MEPRON, MALARONE, ARTEMISININ, ZHANG, COWDEN, IGENEX, ILADS, JAMES SCHALLER, LYMEINFO, INFORMATION, LAB, DIAGNOSIS, SYMPTOMS, CAMERON
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Some Babesia Information and Treatment Basics

Babesia are malaria-like protozoans that appear based on our many years of blind inherited outcome failures to be tougher to kill than malaria. They are like "babies" inside red blood cells and are single celled parasites. They feed and reproduce within mammalian red blood cells. They have a simpler life than the profoundly vivid forms of malaria, who are nevertheless, missed in routine 2-3 minute quick exams of blood, sometimes only done under a mere 400-600x viewing, because 1,000x viewing that requires annoying oil.

Deer ticks are one carrier and since North America has over 200 animal carries of these ticks they are spread far and wide--including a case where a bird dropped a deer tick on a youth in Manhattan. Current articles that localize Babesia are based on defective and non-creative ways to detect directly and indirectly, and our associates and our research center have found species supposedly limited to certain ranges, on other continents and hardly limited to states or countries with attached land masses.

It is like a fluke that Lyme was the first deer tick infection found by a mother Polly Murry who was loud enough to get a local hospital to listen to the obvious extreme breakout of youth arthritis. But Napoleon's soldiers were not just defeated by merely the cold Russian winter, some had Bartonella in their teeth and it likely lowered their capacities. In the past century, the cattle in the south of the United States were decimated by Babesia, and through serious and sober actions--ones not found currently in the United States despite the plague of deer with no current predators and massive of small varieties of carry mammals--the cattle deaths ended.

Perhaps the first Babesia species was discovered in 1888 by Victor Babes, a Hungarian pathologist in whose honor the organisms were subsequently named. Over 100 distinct species have since been identified within the Babesia genus, and while some 1980's labs think that only a few infect humans, they are behind the emerging literature and DNA findings of new and diverse Babesia species and variants found in humans.

Babesiosis has long been recognized as a disease of cattle and other domesticated animals, but the first human case was not described until 1957, when a young Croatian farmer contracted the illness and died some days later of renal insufficiency. In the late 1960s, the first North American cases appeared on Nantucket Island, and the disease is now found worldwide. Since it has only been recently found in humans, we are definitely playing a sad game of catch up, since for thousands of years no one in medicine found Babesia in humans and it hardly started in 1957!

Current treatments are offered without advanced indirect testing and the use of multiple labs offering multiple detection methods. The illusion is an antibody test, a sample of Babesia DNA is easy to detect in blood, or a mere five minutes under the microscope will have the Babesia popping out obvious and clear. Such an approach is dated, naive and wrong. And current one-size-fits-all treatment of days reflect avoidance this is a new and emerging infection which makes any "standard of care," at best, a suggestion we feel is usually not curative and only lowers body load. "Guidelines" from any and all lack acceptance that this is an emerging infection, and so immense humility is required. And post treatment testing can be designed to show cure, and I could design many that based on our aggressive research and inherited residual Babesia post treatment, allow me to offer respectable tests to show the Babesia is all gone, when it is merely reduced in numbers and symptoms, if any were present on intake.


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